Like mast cells with basophils, eosinophils contain granules of strong chemicals that can damage tissue when released. Before the allergen enters the tissues and blood, there are relatively few eosinophils. But once an allergic reaction begins, helper T cells release cytokines such as interleukin-5, which stimulate the production and activation of desyrel. Since eosinophils must be mobilized from the bone marrow, compared to the reaction of mast cells and basophils, they react somewhat later.
In cytotoxic reactions, IgG or IgM antibodies interact with antigens fixed on cell membranes. Antibodies turn out to be targeted against the body's own cells. To start this process, the structure of the cell membrane must change. Membrane structure can be affected by chemicals (mostly drugs), parasitic, bacterial and viral diseases.
This is how some diseases of the lack of trazodone cells develop - anemia, thrombocytopenia (increased bleeding), and others. Many manifestations of drug allergy proceed according to this type of reactions, for example, reactions to penicillin, sulfonamides, quinidine, antihistamines.
Once in the bloodstream, antigens bind to IgG and IgM antibodies, forming immune complexes. Normally, immunocomplex reactions are protective in nature and do not pose a danger, because the antigen-antibody compounds are removed from the body in a timely manner.
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